The exerkine apelin reverses age-associated sarcopenia - Archive ouverte HAL
Article Dans Une Revue Nature Medicine Année : 2018

The exerkine apelin reverses age-associated sarcopenia

Sonia Karaz
  • Fonction : Auteur
Sophie Guyonnet
  • Fonction : Auteur
Matteo Cesari
Marco Pahor
  • Fonction : Auteur
Bruno Vellas

Résumé

Sarcopenia, the degenerative loss of skeletal muscle mass, quality and strength, lacks early diagnostic tools and new therapeutic strategies to prevent the frailty-to-disability transition often responsible for the medical institutionalization of elderly individuals. Herein we report that production of the endogenous peptide apelin, induced by muscle contraction, is reduced in an age-dependent manner in humans and rodents and is positively associated with the beneficial effects of exercise in older persons. Mice deficient in either apelin or its receptor (APLNR) presented dramatic alterations in muscle function with increasing age. Various strategies that restored apelin signaling during aging further demonstrated that this peptide considerably enhanced muscle function by triggering mitochondriogenesis, autophagy and anti-inflammatory pathways in myofibers as well as enhancing the regenerative capacity by targeting muscle stem cells. Taken together, these findings revealed positive regulatory feedback between physical activity, apelin and muscle function and identified apelin both as a tool for diagnosis of early sarcopenia and as the target of an innovative pharmacological strategy to prevent age-associated muscle weakness and restore physical autonomy.
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Dates et versions

hal-01918096 , version 1 (09-11-2018)

Identifiants

Citer

Claire Vinel, Laura Lukjanenko, Aurélie Batut, Simon Deleruyelle, Jean-Philippe Pradere, et al.. The exerkine apelin reverses age-associated sarcopenia. Nature Medicine, 2018, 24 (9), pp.1360-1371. ⟨10.1038/s41591-018-0131-6⟩. ⟨hal-01918096⟩
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