Predicting and Tracking Short Term Disease Progression in Amnestic Mild Cognitive Impairment Patients with Prodromal Alzheimer’s Disease: Structural Brain Biomarkers - Archive ouverte HAL
Article Dans Une Revue Journal of Alzheimer's Disease Année : 2019

Predicting and Tracking Short Term Disease Progression in Amnestic Mild Cognitive Impairment Patients with Prodromal Alzheimer’s Disease: Structural Brain Biomarkers

1 Istituto Centro San Giovanni di Dio Fatebenefratelli
2 UNITN - Università degli Studi di Trento = University of Trento
3 IRCCS - Istituto di Ricerche Farmacologiche "Mario Negri" = Mario Negri Institute for Pharmacological Research
4 UNIROMA - Università degli Studi di Roma "La Sapienza" = Sapienza University [Rome]
5 San Raffaele Pisana IRCCS
6 INS - Institut de Neurosciences des Systèmes
7 TIMONE - Hôpital de la Timone [CHU - APHM]
8 Leipzig University / Universität Leipzig
9 IDIBAPS - Institut d'Investigacions Biomèdiques August Pi i Sunyer
10 UniGe - Università degli studi di Genova = University of Genoa
11 UNIPG - Università degli Studi di Perugia = University of Perugia
12 ICHN - Imagerie cérébrale et handicaps neurologiques
13 UniBs - Università degli Studi di Brescia = University of Brescia
14 Unicatt - Università cattolica del Sacro Cuore = Catholic University of the Sacred Heart [Roma]
15 SDN - Istituto di Ricerca Diagnostica e Nucleare
16 Aristotle University of Thessaloniki
17 Amsterdam UMC - Amsterdam University Medical Centers
18 Universität Duisburg-Essen = University of Duisburg-Essen [Essen]
19 Georg-August-University = Georg-August-Universität Göttingen
20 Universidade de Aveiro
21 GSK - GlaxoSmithKline [Stevenage, UK]
22 CHRU Lille - Centre Hospitalier Régional Universitaire [CHU Lille]
23 TCDV - Troubles cognitifs dégénératifs et vasculaires - U 1171 - EA 1046
24 INT - Institut de Neurosciences de la Timone
25 APHM - Assistance Publique - Hôpitaux de Marseille
26 UNIGE - Université de Genève = University of Geneva

Résumé

Background: Early Alzheimer's disease (AD) detection using cerebrospinal fluid (CSF) biomarkers has been recommended as enrichment strategy for trials involving mild cognitive impairment (MCI) patients. Objective: To model a prodromal AD trial for identifying MRI structural biomarkers to improve subject selection and to be used as surrogate outcomes of disease progression. Methods: APOE ɛ4 specific CSF Aβ42/P-tau cut-offs were used to identify MCI with prodromal AD (Aβ42/P-tau positive) in the WP5-PharmaCog (E-ADNI) cohort. Linear mixed models were performed 1) with baseline structural biomarker, time, and biomarker×time interaction as factors to predict longitudinal changes in ADAS-cog13, 2) with Aβ42/P-tau status, time, and Aβ42/P-tau status×time interaction as factors to explain the longitudinal changes in MRI measures, and 3) to compute sample size estimation for a trial implemented with the selected biomarkers. Results: Only baseline lateral ventricle volume was able to identify a subgroup of prodromal AD patients who declined faster (interaction, p = 0.003). Lateral ventricle volume and medial temporal lobe measures were the biomarkers most sensitive to disease progression (interaction, p≤0.042). Enrichment through ventricular volume reduced the sample size that a clinical trial would require from 13 to 76%, depending on structural outcome variable. The biomarker needing the lowest sample size was the hippocampal subfield GC-ML-DG (granule cells of molecular layer of the dentate gyrus) (n = 82 per arm to demonstrate a 20% atrophy reduction). Conclusion: MRI structural biomarkers can enrich prodromal AD with fast progressors and significantly decrease group size in clinical trials of disease modifying drugs.

Dates et versions

hal-03623863 , version 1 (29-03-2022)

Identifiants

Citer

Moira Marizzoni, Clarissa Ferrari, Jorge Jovicich, Diego Albani, Claudio Babiloni, et al.. Predicting and Tracking Short Term Disease Progression in Amnestic Mild Cognitive Impairment Patients with Prodromal Alzheimer’s Disease: Structural Brain Biomarkers. Journal of Alzheimer's Disease, 2019, 69 (1), pp.3-14. ⟨10.3233/JAD-180152⟩. ⟨hal-03623863⟩
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