N‐3 PUFA deficiency disrupts oligodendrocyte maturation and myelin integrity during brain development - Archive ouverte HAL
Article Dans Une Revue Glia Année : 2022

N‐3 PUFA deficiency disrupts oligodendrocyte maturation and myelin integrity during brain development

Corinne Joffre
Sophie Layé

Résumé

Westernization of dietary habits has led to a progressive reduction in dietary intake of n-3 polyunsaturated fatty acids (n-3 PUFAs). Low maternal intake of n-3 PUFAs has been linked to neurodevelopmental disorders, conditions in which myelination processes are abnormal, leading to defects in brain functional connectivity. Only little is known about the role of n-3 PUFAs in oligodendrocyte physiology and white matter development. Here, we show that lifelong n-3 PUFA deficiency disrupts oligodendrocytes maturation and myelination processes during the postnatal period in mice. This has long-term deleterious consequences on white matter organization and hippocampus-prefrontal functional connectivity in adults, associated with cognitive and emotional disorders. Promoting developmental myelination with clemastine, a first-generation histamine antagonist and enhancer of oligodendrocyte precursor cell differentiation, rescues memory deficits in n-3 PUFA deficient animals. Our findings identify a novel mechanism through which n-3 PUFA deficiency alters brain functions by disrupting oligodendrocyte maturation and brain myelination during the neurodevelopmental period.
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Dates et versions

hal-03347586 , version 1 (17-09-2021)

Identifiants

Citer

Quentin Leyrolle, Fanny Decoeur, Cyril Dejean, Marie-Galadriel Briere, Stephane Leon, et al.. N‐3 PUFA deficiency disrupts oligodendrocyte maturation and myelin integrity during brain development. Glia, 2022, 70 (1), pp.50-70. ⟨10.1002/glia.24088⟩. ⟨hal-03347586⟩
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