Functional characterization of the 12p12.1 renal cancer-susceptibility locus implicates BHLHE41 - Archive ouverte HAL
Article Dans Une Revue Nature Communications Année : 2016

Functional characterization of the 12p12.1 renal cancer-susceptibility locus implicates BHLHE41

Leandro Colli
  • Fonction : Auteur
Mitchell Machiela
  • Fonction : Auteur
Lea Jessop
  • Fonction : Auteur
Timothy Myers
  • Fonction : Auteur
Julie Carrouget
  • Fonction : Auteur
Sarah Wagner
  • Fonction : Auteur
David Roberson
  • Fonction : Auteur
Caroline Eymerit
  • Fonction : Auteur
Stephen Chanock

Résumé

Genome-wide association studies have identified multiple renal cell carcinoma (RCC) susceptibility loci. Here, we use regional imputation and bioinformatics analysis of the 12p12.1 locus to identify the single-nucleotide polymorphism (SNP) rs7132434 as a potential functional variant. Luciferase assays demonstrate allele-specific regulatory activity and, together with data from electromobility shift assays, suggest allele-specific differences at rs7132434 for AP-1 transcription factor binding. In an analysis of The Cancer Genome Atlas data, SNPs highly correlated with rs7132434 show allele-specific differences in BHLHE41 expression (trend P value=6.3 × 10(-7)). Cells overexpressing BHLHE41 produce larger mouse xenograft tumours, while RNA-seq analysis reveals that constitutively increased BHLHE41 induces expression of IL-11. We conclude that the RCC risk allele at 12p12.1 maps to rs7132434, a functional variant in an enhancer that upregulates BHLHE41 expression which, in turn, induces IL-11, a member of the IL-6 cytokine family

Dates et versions

hal-01396749 , version 1 (14-11-2016)

Identifiants

Citer

Pierre Bigot, Leandro Colli, Mitchell Machiela, Lea Jessop, Timothy Myers, et al.. Functional characterization of the 12p12.1 renal cancer-susceptibility locus implicates BHLHE41. Nature Communications, 2016, 7 (12098), pp.1-10. ⟨10.1038/ncomms12098⟩. ⟨hal-01396749⟩

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