Olmesartan-associated enteropathy: results of a national survey. - Archive ouverte HAL
Article Dans Une Revue Alimentary Pharmacology and Therapeutics Année : 2014

Olmesartan-associated enteropathy: results of a national survey.

L. Marthey
  • Fonction : Auteur
P. Pouderoux
  • Fonction : Auteur
J. Lacroute
  • Fonction : Auteur
F. Skinazi
  • Fonction : Auteur
B. Mesnard
  • Fonction : Auteur
J. A. Chayvialle
  • Fonction : Auteur
A. Druez
  • Fonction : Auteur
D. Parlier
  • Fonction : Auteur
M. Gompel
  • Fonction : Auteur
M. Eoche
  • Fonction : Auteur
E. Poncin
  • Fonction : Auteur
R. Bobichon
  • Fonction : Auteur
P. Colardelle
  • Fonction : Auteur
H. Salloum
  • Fonction : Auteur
S. Peschard
  • Fonction : Auteur
F. Zerbib
  • Fonction : Auteur
B. Méresse
  • Fonction : Auteur
N. Cerf-Bensussan
  • Fonction : Auteur
G. Malamut
  • Fonction : Auteur

Résumé

BACKGROUND: Recently, a new enteropathy has been described: olmesartan-associated enteropathy. However, the association has been questioned: a phase 3 trial and a cohort study found no association between gastrointestinal events and olmesartan. AIM: We collected French cases of sartan-associated enteropathy to describe further this entity, confirm or refute causality, and determine if the association exists with other sartans. METHODS: French gastroenterologists were invited to report cases of sartan-associated enteropathy and collect clinical, biological and histological data. Patients with diarrhoea and histological duodenal abnormalities were included. RESULTS: Thirty-six patients with olmesartan-associated enteropathy were reported, including 32 with villous atrophy and four without. There was only one patient with irbesartan-associated enteropathy. None of the patients died. Patients with villous atrophy had diarrhoea, vomiting, renal failure, hypokalaemia, body weight loss and hypoalbuminaemia. Thirty-one patients were hospitalised; four required intensive care. Anti-transglutaminase and anti-enterocyte antibodies were negative; anti-nuclear antibodies were positive (9/11). Endoscopic duodenal biopsies showed villous atrophy (32/32) and polyclonal intra-epithelial CD3+CD8+ lymphocytosis (11/11). Exactly, 14/15 patients responded to steroids and/or immunosuppressants, prescribed because of suspected autoimmune enteropathy. Ten olmesartan interruptions were followed by reintroductions before steroids or immunosuppressants. Interruptions were followed by remissions (9/10), but reintroductions were followed by relapses (9/9). Twenty-nine patients were in remission since olmesartan interruption, including 26 without immunosuppressants. Patients with normal villi had similar clinical characteristics, but mild histological abnormalities (intra-epithelial lymphocytosis and lamina propria lymphocytic infiltration). CONCLUSIONS: Olmesartan causes a severe and immune-mediated enteropathy, with or without villous atrophy. Enteropathy associated with other sartans seems to be very rare.

Domaines

Chimie organique

Dates et versions

hal-01064606 , version 1 (16-09-2014)

Identifiants

Citer

L. Marthey, G. Cadiot, P. Seksik, P. Pouderoux, J. Lacroute, et al.. Olmesartan-associated enteropathy: results of a national survey.. Alimentary Pharmacology and Therapeutics, 2014, 40 (9), pp.1103-1109. ⟨10.1111/apt.12937⟩. ⟨hal-01064606⟩
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