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            <title xml:lang="en">Histomorphological study of the preputial and clitoral glands in BALB/c mice with experimental Taenia crassiceps infections.</title>
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              <classCode scheme="mesh">Animals</classCode>
              <classCode scheme="mesh">Atrophy</classCode>
              <classCode scheme="mesh">Sebaceous Glands</classCode>
              <classCode scheme="mesh">Sex Behavior, Animal</classCode>
              <classCode scheme="mesh">Sex Factors</classCode>
              <classCode scheme="mesh">Specific Pathogen-Free Organisms</classCode>
              <classCode scheme="mesh">Taeniasis</classCode>
              <classCode scheme="mesh">Time Factors</classCode>
              <classCode scheme="mesh">Clitoris</classCode>
              <classCode scheme="mesh">Comparative Study</classCode>
              <classCode scheme="mesh">Female</classCode>
              <classCode scheme="mesh">Lymphocytes</classCode>
              <classCode scheme="mesh">Male</classCode>
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              <p>Male preputial and female clitoral glands of mice undergo development that depends on the level of hormones in the animal. Experimental infection with Taenia crassiceps cysticerci results in significant physiological modifications in the host. Here, we investigated the histomorphological alterations induced by the parasite in these pheromonal glands. Preputial and clitoral glands were recovered from mice at 15, 35, 50, and 70 days postinfection (DPI). The glands were examined macroscopically and microscopically after histological preparation. Male preputial glands show a marked atrophy 35 days after infection. This atrophy is the result of a disorganization of the acinus tissue structure. During the course of infection, the basal, intermediate, and mature acinar cell layers are reduced, and finally, at 70 DPI, the gland includes only the duct system and fibrotic structures. In contrast, females are not affected by the infection because no modifications were observed in the morphology or histology of the clitoral glands. A probable cause for such a divergence between infected male and female mice might be related to a sex steroid imbalance as described during T. crassiceps infection.</p>
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